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The effect of the decrease of the serum prolactin levels on prolactin receptor in the rat testis was studied during rat spermatogenesis using Western blot analysis. After pituitary microsurgery, the number of the serum prolactin (PRL) receptors increased on day 11. This increase was accompanied by an increase in the number of spermatogenic cells on days 15-17. In contrast, a decrease in the number of PRL receptors in the testis was observed in animals with chronic hyperprolactinemia induced by pituitary stalk section (P-SS) or replacement treatment (PRT). Serum PRL levels were decreased and serum testosterone levels were increased in P-SS animals on day 13, which was the same time at which the number of spermatogenic cells was decreased. On day 14, a slight increase in the number of PRL receptors was observed in P-SS animals, which was related to the increased number of spermatogenic cells. These results show that during normal spermatogenesis, PRL receptors are produced on preleptotene spermatocytes, and the number of PRL receptors is increased at the spermatogenic cell stage. Chronic hyperprolactinemia inhibits the production of PRL receptors and thereby inhibits the mitosis of spermatocytes. This may result in spermatogonia loss. These results also suggest that the increase in serum testosterone induced by PRT may be responsible for the increase of the PRL receptors during spermatogenesis.Sunwapta River
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